Editorial: in praise of cognitive neuropsychiatry.

نویسنده

Chris Frith

چکیده

I wonder if the readers of this journal remember that time, not so long ago, when functional psychoses such as schizophrenia had nothing to do with the brain? This belief was not limited to antipsychiatrists such as Szasz. When my colleague, psychiatrist Eve Johnstone, published her classic paper in 1976 in the Lancet demonstrating enlarged ventricles in association with chronic schizophrenia (Johnstone, Frith, Crow, Husband, & Kreel, 1976), the UK’s leading neurologist wrote a letter to the journal suggesting that this enlargement was due to chronic treatment with neuroleptic drugs (Marsden, 1976). This position that the brain had nothing to do with schizophrenia had to be abandoned, not only because of the many replications demonstrating structural changes (see Steen, Mull, McClure, Hamer, & Lieberman, 2006, for a recent review), but also because of robust demonstrations of a role for dopamine in the production of positive symptoms such as hallucinations and delusions (Johnstone, Crow, Frith, Carney, & Price, 1978; Seeman & Lee, 1975). Such observations led to exciting questions about how abnormalities in the physical activity of a brain can lead to abnormalities in subjective experience. This problem was put in stark form when a

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